Statement:
In recent
years there has been increasing public awareness of transvestism and transsexuality.
The media press, radio, television, (with programmes such as Gender
Change and reality T.V. shows) and in particular the
world wide web – are portraying these behaviours as if they were an unavoidable,
are therefore a normal and acceptable, variant of human sexuality.
The message being conveyed is that an enlightened
society ought to be accepting of such people as they present – as a matter
of human right.
In a post modern, ideological driven, era
the church is being challenged as never before to develop an appropriate
Christian response in matters of human sexuality and confused gender identity.
Special interest groups such as Press
for Change, GIRES and others have relied on specifically selected
research documents, often without peer review, from scientific publications
which they (not necessarily the authors) claim demonstrate an innate or
biological determined medical condition which enforces the behaviour –
known clinically as Gender Identity Disorder (GID) or – transsexuality.
Of concern is that these same special interest
groups promote deliberately misleading information in their literature,
and on their web sites, to members of the public and others. (Of particular
concern is that such information was widely circulated to politicians
in an endeavour to influence them (successfully) to support the then Gender
Recognition Bill.)
Vulnerable people visit such web sites
and read the literature. Some, possibly already having a disposition to
transvestism and transsexuality, can be strongly influenced to believe
that the only satisfactory outcome for them is to assume a life – possibly
supported by medical intervention – in their preferred self assumed gender
role.
The material on
the web site is misleading. Although saying the cause of transgender
is not known, they present much material which supports to some extent
a biological link, and by implication an inescapable progression, while
ignoring other interpretations which are more worthy of credence. I
note that they ignore the concept of a mistaken idea in a young person,
reinforced thousands of times by repeated dwelling on the subject leading
to an entrenched idea by adulthood. The authors even present some relevant
literature to support this latter idea but do not comment on it.
They give an
incorrect interpretation of twin studies, which correctly interpreted
show that their implication about overwhelming biological origins is
wrong.
They correctly
state that the transgender concordance in identical twins is low, but
then argue this shows some presumably strong genetic influence. Not
at all. A low concordance means either that genetic (i.e. biological)
influence is low, or that environmental effects can counteract genetic
influence. This means in fact that their entire biological argumentation
is wrong, in so far as it implies a strong influence.
The correct interpretation
of the twin studies is that factors other than genetic are strong, particularly
random factors. An example of a random factor could be one twin but
not the other being told by a relative that if he persists in girl-like
behaviour he might grow up as a girl. This could permanently influence
him, but not his brother who was in another corner of the room.
The authors note
correctly that many children have GID but only in a very small number
of cases do they grow up to become transgendered. This argues that there
is much change going on at early ages (in fact it is known from child
development studies that gender is only thought unchangeable from about
the age of 5-6 on) and GID is not fixed for all time. It argues there
is a rather rare thought pattern which gets fixed in the mind, specifically
that he is a woman in a man's body.
The material
on biological origins is highly speculative, but that children take
strange ideas into their heads and sometimes let them ferment for years
is so well known that it hardly needs scientific support.
N.E.W.
At least one medical clinic in the U.K.
has openly advertised on the web and has run newspaper advertisements
appealing to people confused about their sex and gender identity offering
hormone treatment and surgery.
It is possible to buy hormones over the
internet without prescription.
Instances are emerging in the U.K., Australia
and the U.S. of individuals who have come to regret sex re-assignment
surgery and are seeking legal redress from both the doctors and hospitals
involved as they struggle to resume their own original biological and
gender identity. There are sufficient numbers such of people, once diagnosed
with Gender Identity Disorder, to demonstrate sufficiently that the condition
is not immutable and therefore predominantly psychological and can be
overcome where sufficient motivation is present.
In spite of this, any challenge to the
entrenched position of these same special interest groups will be met
with vociferous accusations of bigotry, narrow mindedness and an unwillingness
to listen to their reasoning and adopt it without question.
Two hypotheses most commonly referred to
by these special interest groups are:
1) That the behaviour is determined in
a localised area of the brain known as the hypothalamus, and
2) That the behaviour occurs as a result
of abnormal hormone surges whilst the fetus still remains unborn in
its mothers womb.
Both these hypothesis are highly speculative
and are being challenged by mainstream medicine. (See
appendix)
In addition, the cortex area of the brain,
which separates animals from humans (animals do not have a cortex.) has
an overriding or executive function which can, if there is
sufficient motivation, override behaviours that that have been repeated
to the point of becoming overwhelming or fixed.
In any event, if any biological pre disposition
can be satisfactorily demonstrated to exist it is likely only to be weak.
Transsexuality in reality arises from a complex variety of circumstances,
predominantly psychological in origin.
These circumstances are likely to have
caused immense personal (psychological) pain. Attempts to avoid that pain
may eventually result in the development of transsexuality. The transsexual
behaviours are pursued defensively as a way inserting themselves into
a fantasy existence – with associated perceived social advantages – as
a way of anaesthetising serious personal pain.
Problems inevitably arise when transsexuality
emerges in a church congregation. With the advent of the Gender Recognition
Act and the associated complications, an appropriate Christian response
has become immensely more difficult. The self determined human rights
of transsexuals are now seen to have priority in determining how Christians
should think and act whenever transsexuality is encountered.
For many churches, issues of salvation,
baptism and communion will be of immense importance. For the pre or post
operative transsexual the question of marriage is also likely be important.
Or, even for someone who simply insists on their perceived human right
to present and be accepted in their own self determined gender role without
having undergone any chemical or surgical intervention.
Notwithstanding the intent of the Gender
Recognition Act, there will be serious expressions of concerns in congregations
as to the effect that the presence of an all too frequently obvious, transsexual
will have, particularly on children. Congregations are similarly sensitive
to the use, by transgendered persons, of facilities such as toilets. Concerns
are expressed when young children, as is common, use designated womens
toilets – not necessarily accompanied by their mothers.
Most people take for granted that their
gender identity and their sexual identity are entirely congruent, that
is – consistent. It is therefore often difficult for them to imagine how
much personal pain can result in the significant skewing of a gender identity
away from biological identity, and, to understand the often obsessive
behaviour associated with it.
The roots of transsexuality, though obscure,
are considered as being multi-factorial, are most likely to have arisen
in response to painful early childhood experiences. A large body of psychiatric
and psychological evidence points to the development of gender identity
occurring below five years of age.
It is common for transgendered persons
to assert that they can recall, earlier than at three years of age, thoughts
that they had of: being born into the wrong body. Psychological
studies evidence that such alleged memories are unreliable.
The limited amount of psychological research
done into the development of gender identity disorder would suggest that
family dysfunction and an inappropriate individual response, are strongly
contributing factors.
Given that, church leaders and congregations
which are concerned with wholeness in Christ will seek more understanding.
Transsexuality, therefore for Christians, needs to be seen as an issue
of personal identity in crisis.
The apostle Paul reminds us that all fall
short of the glory of God. The Bible indicates that all have deviated
in some way from Gods original creative intent. Christians are fallen
creatures, who are themselves wounded healers, and ought to be about pursuing
wholeness in Christ.
Biblically-based personal wholeness will
come about in a measure consistent with ones willingness to be obedient
to the Lordship of Christ in ones own life.
Adherence to a biblical statute alone will
not bring about personal wholeness. Wholeness is not about putting the
past behind and ignoring personal pain. All pain is an indicator that
something is not as it should be.
Personal pain needs to be seen as the basis
from which personal growth and wholeness can develop within a supportive
Christian community. Achieving personal growth into the person God planned
them to be will be slow, and in itself painful.
For the person with a background of confused
gender identity, as further pain is encountered seeking after biblical
wholeness, or unexpected crisis occurs, there will be a tendency to resort
to former patterns of behaviour. Pain threshold is likely to be low. If
despair sets in it is likely to result in an unwillingness to co-operate
further in seeking after wholeness in Christ.
Old patterns of behaviour will be cyclical
and can be identified. If recognised, and there is sufficient will by
all parties concerned to do so, new patterns of behaviour can be established
and maintained.
For post operative male to female transsexuals
there will need to be an acceptance that surgically removed genitalia
cannot be satisfactorily replaced.
Expectation of acceptance into the assumed
gender role by members of the biological sex is likely to be limited increasing
a sense of isolation and alienation from society.
Pastoral oversight of gender confused patients
will be fraught with difficulties for all concerned.
Jesus himself demonstrated an extraordinary
willingness to meet people where they are at. In the third
chapter of Johns gospel we see a picture of Jesus meeting with a
Pharisee – a well-educated religious leader. In the next chapter we see
Him meeting with a Samaritan woman Samaritans being despised by
the Jews. In the eighth chapter we see Jesus meeting with someone legally
condemned by the law for adultery. Rather than run from Jesus they appeared
to be drawn to Him. We need to look to His example to guide us in a pastoral
response to the gender confused.
Appendix 1.
It really deals a
death blow to any attempt to say that that structure can be responsible
for childhood feelings of being the wrong gender
. N
E Whitehead Ph D
In Humans, the Central Subdivision of the Bed Nucleus
of the Stria Terminalis Does Not Become Sexually Dimorphic Until Adulthood
By Anne A. Lawrence, M.D., Ph.D.
--------------------------------------------------------------------------------
Two reports from the Netherlands
Institute for Brain Research, published in 1995 and 2000, have been widely
interpreted as demonstrating a possible neuroanatomic marker for male-to-female
(MtF) transsexuality. In 1995, Zhou et al. reported that the central subdivision
of the bed nucleus of the stria terminalis (BSTc), a collection of cells
in the hypothalamus, was sexually dimorphic in humans. BSTc volume was
significantly greater in male brains than in female brains. However, the
brains of six MtF transsexuals Zhou et al. studied were found to have
BSTc volumes within the typical female range. A follow-up paper by Kruijver
et al. (2000) looked at the same six transsexual brains and two additional
ones, including one from a female-to-male transsexual. Kruijver et al.
reported that the sexual dimorphism in BSTc volume found by Zhou et al.
reflected a genuine difference in neuron number, and was not simply an
artifact of the measurement technique Zhou et al. used.
These two reports have spawned
some truly fantastic extrapolations, including the assertion by some MtF
transsexuals that they have the "brain-sex" of females (example:
"[I] was born and raised as a boy. It was a terrible mistake,
because [I] had the brain-sex . . . of a girl."). Unqualified
assertions like this have been made even by persons trained as scientists.
Never mind that the papers cited above studied only eight transsexual
brains, and have never been replicated in another laboratory. Never mind
that, of the dozens of other sexually dimorphic structures in the
human brain, not one has been shown to be sex-atypical in transsexuals.
Never mind that we have no idea exactly what functions the BSTc might
subserve in humans. The true believers in transsexual "brain-sex"
had found their biologic marker, and no amount of ordinary scientific
skepticism could dissuade them.
Until, perhaps, now. A recent paper
by Chung et al. (2002) has demonstrated a most unexpected finding: In
humans, unlike the rat and perhaps other species, BSTc volume does not
become sexually dimorphic until well into adulthood. However, most MtF
transsexuals report that they experienced gender dysphoria beginning in
childhood, often from the time of their earliest memories. It is hard
to imagine how BSTc volume could be a marker for gender identity if BSTc
volume has not yet become sexually dimorphic at a time when gender identity
has already been firmly established. As Chung et al. wrote:
"Late sexual differentiation
of the human BSTc volume also affects our perception about the relationship
between BSTs [sic] volume and transsexuality. Interestingly, transsexuals
receive their first consultation between the ages of 20 and 45 years,
which coincides with the period of sex-dependent divergence of BSTc volume
found in the present study (Van Kesteren et al., 1996). However, epidemiological
studies show that the awareness of gender problems is generally present
much earlier. Indeed, [about] 67-78% of transsexuals in adulthood report
having strong feelings of being born in the wrong body from childhood
onward (Van Kesteren et al., 1996)."
Of course, it is still possible
to construct explanations of these findings that are consistent with the
hypothesis that BSTc volume in adulthood could be a marker for gender
identity in MtF transsexuals. Chung et al. conjectured that fetal or neonatal
hormone levels could affect gender identity, and could perhaps simultaneously
produce changes in BSTc "synaptic density, neuronal activity, or
neurochemical content" that might not immediately affect BSTc volume,
but that might somehow do so much later, during adulthood. Naturally,
they did not propose any specific mechanisms by which such delayed effects
might occur. Alternatively, they conjectured, failure to develop a male
gender identity might itself somehow affect adult BSTc volume, again by
an unspecified mechanism.
These possibilities notwithstanding,
it should be apparent that the Chung et al. paper has dealt a serious
blow to the hypothesis that BSTc volume is a biologic marker for gender
identity in MtF transsexuals. Whether this will diminish the fanciful
claims of "female brain-sex" by MtF transsexuals themselves
remains to be seen.
Here is the complete citation and abstract for the Chung et al. paper:
Sexual differentiation
of the bed nucleus of the stria terminalis in humans may extend into adulthood.
Journal of Neuroscience 22(3): 1027-1033 (February 1, 2002)
Chung WC, De Vries GJ, Swaab DF.
Netherlands Institute for Brain Research, 1105 AZ Amsterdam, The Netherlands.
ABSTRACT: Gonadal steroids
have remarkable developmental effects on sex-dependent brain organization
and behavior in animals. Presumably, fetal or neonatal gonadal steroids
are also responsible for sexual differentiation of the human brain. A
limbic structure of special interest in this regard is the sexually dimorphic
central subdivision of the bed nucleus of the stria terminalis (BSTc),
because its size has been related to the gender identity disorder transsexuality.
To determine at what age the BSTc becomes sexually dimorphic, the BSTc
volume in males and females was studied from midgestation into adulthood.
Using vasoactive intestinal polypeptide and somatostatin immunocytochemical
staining as markers, we found that the BSTc was larger and contains more
neurons in men than in women. However, this difference became significant
only in adulthood, showing that sexual differentiation of the human brain
may extend into the adulthood. The unexpectedly late sexual differentiation
of the BSTc is discussed in relation to sex differences in developmental,
adolescent, and adult gonadal steroid levels.
References:
Chung WC, De Vries GJ, Swaab DF
(2002) Sexual differentiation of the bed nucleus of the stria terminalis
in humans may extend into adulthood. J Neurosci 22(3): 1027-1033.
Kruijver FP, Zhou JN, Pool CW,
Hofman MA, Gooren LJ, Swaab DF (2000) Male-to-female transsexuals have
female neuron numbers in a limbic nucleus. J Clin Endocrinol Metab
85(5): 2034-2041.
Van Kesteren PJ, Gooren LJ, Megens
JA (1996) An epidemiological and demographic study of transsexuals in
the Netherlands. Arch Sex Behav 25(6): 589-600.
Zhou JN, Hofman MA, Gooren LJ,
Swaab DF (1995) A sex difference in the human brain and its relation to
transsexuality. Nature 378(6552): 68-70.
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© 2002 by Anne A. Lawrence,
M.D., Ph.D. All rights reserved.
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